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Nexum (Esomeprazole)
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Esomeprazole

Nexum is used to treat the symptoms of gastroesophageal reflux disease (GERD), a condition in which backward flow of acid from the stomach causes heartburn and possible injury of the esophagus (the tube between the throat and stomach) in adults and children 1 year of age and older. Prescription Nexum is used to treat damage from GERD in adults and children 1 month of age and older. Prescription Nexum is used to allow the esophagus to heal and prevent further damage to the esophagus in adults with GERD. Prescription Nexum is also used to decrease the chance that people who are taking nonsteroidal anti-inflammatory drugs (NSAIDs) will develop ulcers (sores in the lining of the stomach or intestine) in adults. It is also used with other medications to treat and prevent the return of stomach ulcers caused by a certain type of bacteria (H. pylori) in adults. Prescription Nexum is also used to treat conditions in which the stomach produces too much acid such as Zollinger-Ellison syndrome in adults. Nonprescription (over-the-counter) Nexum is used to treat frequent heartburn (heartburn that occurs at least 2 or more days a week) in adults. Nexum is in a class of medications called proton pump inhibitors. It works by decreasing the amount of acid made in the stomach.

Other names for this medication:
Esomeprazole, Axagon, Ceso, Emep, Esofag, Esomac, Esomep, Esomeprazol, Esomezol, Esonix, Esopral, Esoprax, Esoprazol, Esoral, Esoz, Espram, Inexium, Izra, Lucen, Maxpro, Nedox, Neksium, Neptor, Nexe, Nexiam, Nexium, Nexpro, Nexum, Nexx, Opton, Progut, Pronex, Raciper, Sergel, Sompraz, Ulcratex, Zosec,

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We have shown the important role of telomere and telomerase in vascular cell senescence in vitro. We estimate the models in a Bayesian framework using Markov chain Monte Carlo posterior simulations. Outbreaks of foot and mouth disease (FMD), African swine fever (ASF), classical swine fever (CSF) and swine vesicular disease (SVD) can cause significant economic and social costs and severe trade limitations. The identification of a potent steroid sulphatase inhibitor lacking any oestrogenicity, such as 2-methoxy EMATE, should be of considerable value in evaluating the potential of steroid sulphatase inhibition for breast cancer therapy.

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In human aortic smooth muscle cells, neither prevention of SOCS-3 protein induction, using STAT3 or SOCS-3 antisense, nor prevention of SHP-2 expression, also with antisense, ablates desensitization. When Leu-421 (located in the oligomerization domain) was mutated to a glutamate residue, CPS formed an (alphabeta)2 dimer in the presence of ornithine, UMP, or IMP. These data suggest that hyperphosphorylation at serine 199/202 of tau factor is induced by MAP kinase, CDK5, and GSK3, and contributes to ischemic neuronal injury.

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